Japanese researchers at Osaka University have developed an experimental drug that can reduce the death of neurons after paralysis. The GAI-17 drug blocks Gapdh protein, which in certain conditions promotes cells of cells and participates in a number of heavy neurological diseases, including Alzheimer's disease. The work was published in the issue of ISCIENCE.
The team led by Associate Professor Hidemic Neaquima has shown that after the stroke, the use of GAI-17 has significantly reduced the damage to the brain tissue and the strategy. Moreover, the medicine was effective even if six hours after the stroke, this is extremely important, as it is not always possible to start treatment immediately.
In addition, the drug has not caused serious side effects, including heart or vascular disorders, which makes it potentially safe for future use.
The authors note that the braking of Gapdh protein aggregation may become a universal approach to treating various brain diseases related to neurodegeneration. The next step will be the testing of GAI-17 in other pathologies models.
Translation of: Euromedia24.com
